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WHAT IS IT?
Horner syndrome refers to the sympathetic denervation of the face. There are a wide variety of causes that are responsible for this (listed below). A classic triad of unilateral symptoms (ptosis, anhidrosis, and miosis) are associated with this condition.
Causes involve pathology above T1: Brown-Séquard syndrome, lesion in PICA artery (Wallenberg syndrome), cavernous sinus syndrome, pancoast tumor (lung cancer), late-stage syringomyelia, carotid dissection, cluster headaches
WHY IS IT A PROBLEM?
A serious underlying condition (such as a carotid dissection) can manifest as Horner syndrome. The cause must be discovered!
WHAT MAKES US SUSPECT IT?
Ptosis is caused by the loss of innervation of the superior tarsal muscle (this is different then the ptosis in cranial never III palsy, which innervates the levator palpebrae).
Anhidrosis will result in a lack of sweating on the affected side of the face.
Miosis loss of the sympathetic innervation to the eye will allow for the parasmypatheticly innervated constriction muscles to operate unopposed.
Facial rubor: redness/flushing of the affected side might also be observable as well
HOW DO WE CONFIRM A DIAGNOSIS?
Horner syndorme is a clinical diagnosis. Uncovering the underlying pathological cause likely requires imaging of the region to discover what exactly is affecting sympathetic innervation.
HOW DO WE TREAT IT?
This will depend on the underlying pathology.
HOW WELL DO THE PATIENTS DO?
This will depend on the underlying pathology.
WAS THERE A WAY TO PREVENT IT?
This will depend on the underlying pathology.
WHAT ELSE ARE WE WORRIED ABOUT?
This will depend on the underlying pathology.
OTHER HY FACTS?
Lesion in the PICA artery can lead to ipsilateral Horner syndrome
FURTHER READING
Page Updated: 06.04.2016